Pictured: German monkeypox patient whose nose started to ROT because his undiagnosed HIV and syphilis left his immune system ravaged

The damage done by the clot shots is only beginning to come to light. How will the future rectify this willful disaster?

Veering off from my biological scope, I have followed Clif High's web bot data for over 20 years. Clif anticipates a crash in real estate as large numbers of people die off reducing demand. Likewise, the die off reduces the number of people actively, competently doing productive work such that some have to shoulder performing 2 or 3 jobs to keep normal civil society functioning. Many will die at home and not be discovered for an extended period for lack of friends and family aware of their existence. My wife is already encountering that as a police dispatcher. A woman was found deceased for what appears to be over 8 months. The mail carrier raised concern with all of the mail piling up.

I read a short Twitter thread from a woman who has a fairly significant number of Youtube channels in her regular watch queue. She commented that many of her long time favorites are significantly cognitively impaired now compared to a couple years back.

What to do? Be aware that it is occurring. If you're capable of being that "fill-in" that keeps the world going, do it. Polish a broad set of skills to compensate for the people who won't be there to provide those services in the future.

Are the doctors sure he didn’t have undiagnosed syphillis from long ago? Plus, HIV infection ages its infected 14 years (although homosexual sites are saying 5 years now, which I presume means that HIV accelerated aging is becoming well known). HIV accelerated aging means they get dementia, cancers, kidney disease, et al., even if they’re taking antiretrovirals. I also assume it kills HIV infected 14 years early. Homosexual sites rarely mention HIV accelerated death.

HIV’s accelerated aging can cause dementia, or HAND: HIV-associated neurocognitive disorder.

The SARS-CoV-2 spike protein includes gp120 and GAG from the HIV virus. The spike protein is a chimera built from multiple pathogens. Modes of damage include disabling of dsDNA repair from the s1 protein acting in the nucleus to disable BRAC1/BRAC2/p53. Unable to repair DNA damage, the cell becomes dysfunctional over time. The s1 subunit is also a prion capable of generating stable amyloid fibrils. The amyloid fibrils are similar to those found an Alzheimer's. The p53 disabling turns a cell "senescent". It de-differentiates and becomes functionally "illiterate". It just lives on consuming resources and doing nothing useful. There are more pathways of damage. Lots of papers documenting what has been identified. Walter Chesnut has done some very informative dot connecting over the past couple years. Check out his substack "wmcresearch".

Are you a biologist, cuz your response is full of specific biology terms.

Yup. Molecular Biologist. Revelle, UCSD, 1976. Another year of grad school in the microbiology dept at SDSU. Decided not to pursue further as a life of living on grants was not appealing. I've made a good living in the EE/CS world since then, but never lost interest in the field.

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