African scientists baffled by monkeypox cases in Europe, US

I’m sure this has nothing to do with it. I’m sure they learned their lesson when they infected everyone with the SV40 monkey virus that was in the polio vaccine.

My family lined up for the sugar cubes with the OPV at Pearl Harbor Elementary School. Early 1962. It was the batch contaminated with SV40. I didn't have my first cancer until July 1985. My mom passed from lung cancer in 2011, but she did smoke from the mid-40s until 1967.

Chimpanzees are apes, not monkeys-they are different

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Do you know what else is different? Chimpanzees and humans.

"Experts have stressed they do not know if the disease is being spread through sex or other close contact related to sex."

Huh? WTH? Apparently being spread in the lgbtqwerty community by people doing immoral and unatural things. Now, like AIDS, they want to "mainstream" it and make the normal population think that they are at just as much risk, so as to relieve the stigma and blame from the actual culprits. Don't even ask how it spread from the monkey population!

What evidence do you have that either WOULD work for a pox virus? It’s completely different from COVID.

I used ivermectin for COVID, and it seemed to work fine, but there simply is no evidence that ivermectin would work for other viruses. Do you have some, any, evidence to the contrary?

Any?

Even the NIH admits ivermectin anti-viral effects on quite a few types of viruses. I am unable to identify whether the viruses listed include pox viruses.

https://pubmed.ncbi.nlm.nih.gov/32942671/

https://www.nature.com/articles/s41429-020-0336-z

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7251046/

Regarding its role as an antiviral agent, its efficacy has been demonstrated on several viruses, both in vitro and in vivo. Among the many mechanisms by which it performs its function, the most consolidated one sees ivermectin as an inhibitor of nuclear transport mediated by the importin α/β1 heterodimer, responsible for the translocation of various viral species proteins (HIV-1, SV40), indispensable for their replication (Wagstaff et al. 2011; Wagstaff et al. 2012). This inhibition appears to affect a considerable number of RNA viruses (Jans et al. 2019; Caly et al. 2012), such as Dengue Virus 1-4 (DENV) (Tay et al. 2013), West Nile Virus (WNV) (Yang et al. 2020), Venezuelan Equine Encephalitis Virus (VEEV) (Lundberg et al. 2013), and Influenza (Gotz et al. 2016). In addition, ivermectin has been shown to be effective against the Pseudorabies virus (PRV, with a DNA-based genome), both in vitro and in vivo (Lv et al. 2018), using the same mechanism. Caly et al. (Caly et al. 2020) have recently shown that the drug also inhibits the replication of the SARS-CoV-2 virus in vitro, however not clarifying how it occurs. Since the causative agent of COVID-19 is an RNA virus, it can be reasonably expected an interference with the same proteins and the same molecular processes described above.

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