The finding of hypocortisolism is consistent with endocrine dysfunctioning in chronic sexual abuse victims and may have clinical implications with regard to treatment possibilities.
See, the chronic sexual abuse victims had a lower cortisol output (hypocortisolism) as they, sadly, were obviously more acclimatized mentally to such abuse and it wasn't as stressful for them any longer, compared to the "adolescent victims of single sexual trauma" which was still generating stress for them and caused them to produce higher cortisol levels.
Thanks for the link, though you probably didn't intend for it to help me as much as it did.
See, the chronic sexual abuse victims had a lower cortisol output (hypocortisolism) as they, sadly, were obviously more acclimatized mentally to such abuse and it wasn't as stressful for them any longer, compared to the "adolescent victims of single sexual trauma" which was still generating stress for them and caused them to produce higher cortisol levels.
You 'forgot' to include the first sentence of the conclusion, which stated that the study supports the hypothesis that victims of a single sexual trauma experience the same dysregulation as chronic victims. Here is the conclusion in its entirety:
ConclusionsThe results show evidence for a dysregulated HPA-axis in female adolescent victims of single sexual trauma with PTSD. The finding of hypocortisolism is consistent with endocrine dysfunctioning in chronic sexual abuse victims and may have clinical implications with regard to treatment possibilities.
Are you having difficulty comprehending what you are reading, or are you deliberately cherry picking to make the source appear to say the opposite of what it actually does?