Totally great tool..!
Oh, except omit the bottom band.
Uh...and then turn it upside down.
Ya know? Just throw out this pyramid totally.
Posted on 12/26/2019 9:27:41 PM PST by ConservativeMind
No baked potato with butter and sour cream?!!!!
That’s only HALF a life.
PharmEPA
Add bacon, sour cream, cheese and green onion. Then throw away the potato.
Eat more mushrooms. Oyster mushrooms contain a naturally occurring “Lovostatin”, which lowers LDL without the side effects of the synthetics. Lions mane mushrooms have been clinically shown to contain neuroregenerative properties, regular consumption of which is helpful with memory and nerve conduction, as well as clinically proven to lower blood sugar. The compounds in lions mane mushrooms have been shown to be helpful with diabetic nerve pain, fibromyalgia, neuropathy, along with Parkinson’s and Alzheimer’s. (and it tastes like lobster) Chestnut mushrooms (Pholiota adiposa) contains a natural ACE inhibitor, which lowers blood pressure. P. adiposa is prescribed in Japan for mild to moderate hypertension. Shiitake, the most clinically studied and most popular culinary mushroom worldwide are loaded with anti-cancer compounds, along with being one of the best sources for Vitamin D3 and trace elements like copper, zinc, magnesium, manganese and iron. Shiitake also contains anti-inflammatory properties, along with being 24% crude protein. Look around at the cultures who eat a lot of mushrooms and you’ll see much lower incidences of cancer, bowel diseases, neurological diseases and infectious diseases. Fungi are our friends.
If you do that then you need corn chips and chili.
But VLDL is effectively 1/5 the number of your triglycerides, so lower your triglycerides and directly lower your VLDL.
Totally great tool..!
Oh, except omit the bottom band.
Uh...and then turn it upside down.
Ya know? Just throw out this pyramid totally.
It’s awfully hard for me to buy into an article for food consumption that is written by an organization that is selling their diet, the EAT–Lancet benchmark diet, and is funded by ultra liberals the Bill and Melinda Gates Foundation.
rwood
28 posts and no mention of the heart benefits of WEED?
This thread is a total bummer and is bringing me dowwwwnnnnnn.......
Everyone with two brain cells knows that cannabis is going to put BigPharma out of business.
Take a couple of tokes and grab some munchies.
We all gonna die, but remember that weed helps your body to metabolize whatever you eat.
Weed makes you live longer and be happy all the time./Heavy sarcasm alert
I’m borderline High cholesterol with low/normal triglycerides.
I refuse to take statins.
I used to feel tired all the time. I had a lot of muscle aches.
But I feel much better now.
I quit taking statins. Seriously.
If statins are so good why are the side effects so bad?
You make a great point about the weed posts, lately.
My German MD said that my 300 cholesterol level was probably NORMAL for me because of genetic factors, as I have had USs of the carotids and even the major vessels around the heart are clear. Here in the US, they put me on Lipitor and I instantly developed neuropathy, stopped the drug and told them I was never taking another statin drug, regardless. I am trying to re-start the Keto diet and have tried to keep the carbs low during the holidays, as it is almost impossible to hold fast to during this time, but today I am going to start giving it my best shot again.
NorthstarMom: >>I was just prescribed a statin because of inherited high levels of lp(a).
Because I don’t have high cholesterol and my good cholesterol is great, I’m kind of nervous about taking it. When I questioned it as statins don’t lower lp(a) my cardiologist consulted with a cardiologist at Mayo Clinic and they confirmed that it is how they treat those with elevated lp(a).
From what I can tell, the idea is that less ldl means there is less for the lp(a) to cling to.
I’m 45 and don’t look like a typical cardiology patient. It’s all because of genetics. I have a heart scan in 2 weeks to see if/how much blockage there is. My younger brother died instantly from a heart attack this fall leaving me as the only one in my immediate family to not have had a massive heart attack.
While I do have the high lp(a), I’m the only one who limits sugar and bad fats in my diet and have done so for the past 14 years. I was prescribed Lipitor yesterday so haven’t taken it yet. I just don’t know what is the right thing to do.<<
I confess to not having known about LP(a) specifically. I am familiar with APOE gene status, and this site says they are physically associated.
https://www.amgenscience.com/features/10-things-to-know-about-lipoproteina/
Apparently, like APOE proteins, LP(a) comprises part of the cholerterol lipoprotein shell.
From the site:
>>6. There are currently no approved medicines that directly target Lp(a).
Statins, the most widely used type of LDL-lowering drugs, do not reduce Lp(a) and may even cause a slight increase.<<
Given the sometimes-fatal side effects associated with statins you couldn’t get me to swallow one on my death bed.
Most people who refer to avoiding “bad fats” are talking about avoiding saturated fats. It’s an outdated distinction. Saturation merely refers to how hard the fat is. Bile acid breaks down hard fats the same as liquid fats. There is no metabolic distinction.
LP(a) status would be like APOE status in that it determines the ratio of HDL-to-LDL cholesterol particles. Neither type of particle is implicated in coronary artery disease. CLDL (VLDL) particles are the culprits.
As I said above, the problem is glycation, not fats. Glycation is caused by carbohydrate ingestion, not fat ingestion. Trade your carbohydrates for fats.
>>Also, the next step after glycation, is the uptake of Calcium into the atherosclerotic deposits.
This is actually reversible with high dose vitamin K in conjunction with vitamin D. Vitamin K is best absorbed when taken with fat, as it is fat-soluble.<<
Circulating calcium comes from the combination of how hypothalmus-pituitary-adrenal (HPA) axis failures create shortages of adrenal aldosterol, combined with autoimmune processes which inappropriately release cellular calcium into the blood. HPA axis dysfunction is caused by carbohydrate infiltration and contamination of the lower brain through the vagus nerve.
Add bacon, sour cream, cheese and green onion. Then throw away the potato.
didn’t you already say this in Post 23
Bkmrk.
https://en.wikipedia.org/wiki/Dystrophic_calcification
>>Dystrophic calcification (DC) is the calcification occurring in degenerated or necrotic tissue, as in hyalinized scars, degenerated foci in leiomyomas, and caseous nodules. This occurs as a reaction to tissue damage<<
So which is the problem? .....the calcium which collects on damaged artery walls, or the ingested carbohydrates which damage artery walls?
Hmmmmmm?
It ain’t his fault.
An extra helping of bacon bits is irresistible.
Disclaimer: Opinions posted on Free Republic are those of the individual posters and do not necessarily represent the opinion of Free Republic or its management. All materials posted herein are protected by copyright law and the exemption for fair use of copyrighted works.