This is actually reversible with high dose vitamin K in conjunction with vitamin D. Vitamin K is best absorbed when taken with fat, as it is fat-soluble.
>>Also, the next step after glycation, is the uptake of Calcium into the atherosclerotic deposits.
This is actually reversible with high dose vitamin K in conjunction with vitamin D. Vitamin K is best absorbed when taken with fat, as it is fat-soluble.<<
Circulating calcium comes from the combination of how hypothalmus-pituitary-adrenal (HPA) axis failures create shortages of adrenal aldosterol, combined with autoimmune processes which inappropriately release cellular calcium into the blood. HPA axis dysfunction is caused by carbohydrate infiltration and contamination of the lower brain through the vagus nerve.
https://en.wikipedia.org/wiki/Dystrophic_calcification
>>Dystrophic calcification (DC) is the calcification occurring in degenerated or necrotic tissue, as in hyalinized scars, degenerated foci in leiomyomas, and caseous nodules. This occurs as a reaction to tissue damage<<
So which is the problem? .....the calcium which collects on damaged artery walls, or the ingested carbohydrates which damage artery walls?
Hmmmmmm?