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NIDDM does have both a genetic and environmental component, so you are correct that it is not solely linked to body weight. However, the genes that predispose to NIDDM (which are multifactorial and so can't be named with 100% certainty, although the genetic component is still undeniable) tend only to be expressed when an individual is overweight and with high circulating lipids, leading to metabolic syndrome.

Now that all being said, if the genetic tendency to develop insulin resistance is especially strong for an individual, that person can develop NIDDM without being overweight. However, if that person were overweight, they would invariably develop the insulin resistance at a younger age.

At least, that's what my biochemistry professor told me when I was an MS1.

However, the genes that predispose to NIDDM (which are multifactorial and so can't be named with 100% certainty, although the genetic component is still undeniable) tend only to be expressed when an individual is overweight and with high circulating lipids, leading to metabolic syndrome.

This is the theory that I disagree with. I believe that the genes are expressed when exposed to environmental stimuli (IE: carbohydrates), even before the weight gain is apparent. Weight gain is a symptom of the gene expression, not a stimulus of the disease.

And you CANNOT have my blood, vampire!! (I'm fetching the Holy Water now. I'm series.)