I see you're still trying to perpetuate the same, tired myths.
Kathleen J Melanson and others at Rhode Island University reviewed the effects of HFCS and sucrose on circulating levels of glucose, leptin, insulin and ghrelin in a study group of lean women. All four tested substances have been hypothesized to play a role in metabolism and obesity. The study found "no differences in the metabolic effects" of HFCS and sucrose in this short-term study, and called for further similar studies of obese individuals and males. ("Similar effects of high fructose corn syrup and sucrose consumption on circulating levels of glucose, leptin, insulin and ghrelin,"
"A recent study by Martine Perrigue, et al at the University of Washington was presented at the April 2006 meeting of Experimental Biology. ("Hunger and satiety profiles and energy intakes following the ingestion of soft drinks sweetened with sucrose or high fructose corn syrup (HFCS)" Program Abstract # LB433) They concluded:"
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Oops, looks like you're still wrong. Now, back to CSPI with you.
Sucrose is broken down during digestion into fructose and glucose through hydrolysis by the enzyme sucrase, by which the body regulates the rate of sucrose breakdown. Without this regulation mechanism, the body has less control over the rate of sugar absorption into the bloodstream.
Sugar is sucrose. HFCS is not sucrose.