How is that even possible?
The body recognizes the sucrose as food. Sends a “stop eating or drinking” signal to the brain, the mouth.
No, that's not how it works at all. Satiation is mediated in the blood. The mechanisms for satiation are controlled by hormones, not enzymes. Hormones control enzymes.
>>>> One theory is that the Sucrase step influences the amount you consume <<<<
>>How is that even possible?<<
It’s not how other satiation pathways have been shown to work, but it’s plausible (in the sense that an undiscovered island populated by unicorns is plausible, but not likely) that there’s an undiscovered psychological response to flavor, wherein consuming more or less of a substance to acheive a similar sensory reward affects consumption of that substance. Not likely though, and it would tend to explain away that one rat study which is the one scientific study out of dozens that HFCS-phobes care about: the rats got used to receiving a certain level of pleasure (in this case from the sweetness), and when it was removed from their diet, they compensated by consuming greater amounts of food. Since 55 HFCS is formulated to approximate sucrose, this would explain away the rat behavior as unanalogous to the issue at hand. (It’s really odd that female rats did not increase their consumption; maybe they were more concerned with their figure?)
My point is that further study is needed to determine exactly how much worse hfcs is than real sugar, and why.
Study after study is coming out pointing out the various dangers of hfcs.
We know less about the digestive system than we claim to.