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To: Black Agnes

I guess we have drifted the thread so far that I have no idea what your point is.

My point is: there are no definitive published scientific studies that support supplements as doing anything at all.

That point continues to be unmolested.


47 posted on 09/08/2014 8:31:40 PM PDT by freedumb2003 (AGW "Scientific method:" Draw your lines first, then plot your points)
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To: freedumb2003
"That point continues to be unmolested."

Which supplement are you speaking of?

There are many studies that link d3 with benefit. Particularly cancer studies.

There are also studies linking supplementation with melatonin ditto ditto.

etc.

How often do you actually read pubmed for any studies on this?

I read it every single day. You?

Try this one:

http://www.ncbi.nlm.nih.gov/pubmed/25179086

" Oxidative stress plays a role in the pathogenic traits of MS. Melatonin possesses antioxidative properties and regulates circadian rhythms. Several studies have reported that the quality of life is worse in patients with MS than in healthy controls, with a higher prevalence of sleep disturbances, depression and fatigue. The aim of study was to evaluate 5 mg daily melatonin supplementation over 90 days on serum malondialdehyde (MDA) concentration and superoxide dismutase (SOD) activity and its' influence on impact of the quality of life of MS patients. A case-control prospective study was performed on 102 MS patients and 20 controls matched for age and sex. The EDSS, MRI examinations and Multiple Sclerosis Impact Scale (MSIS-29) questionnaire was completed. Marked increase in serum MDA concentration in all MS patients groups was observed and after melatonin treatment decreased significantly in interferons-beta and glatiramer acetate-treated groups, but not in mitoxantrone-treated group. A significant increase in SOD activity compared to controls only in glatiramer acetate-treated group was observed. After 3 months melatonin supplementation the SOD activity increased compared to initial values in interferons beta-treated groups. A significant increase in both MSIS-29-PHYS and MSIS-29-PSYCH items mean scores only in the MX group as compared to other groups was observed. There were no significant differences in mean MSIS-29-PHYS was observed before and after melatonin therapy. Melatonin supplementation caused a decrease in mean MSIS-29-PSYCH scores compared to initial values in interferons beta-treated groups. Finding from our study suggest that melatonin can act as an antioxidant and improves reduced quality in MS patients. "

http://www.ncbi.nlm.nih.gov/pubmed/24628045

"The aim of this study was to evaluate the efficacy of oral melatonin supplementation on oocyte and embryo quality in patients in an assisted reproductive technologies program. All patients were treated for at least 2 weeks with melatonin (3 mg/day). To evaluate the cumulative effect of melatonin supplementation, we compared cycle outcomes between the first (no supplementation) and second cycles (melatonin supplementation) of patients who completed two treatment cycles. There were no significant differences in maturation rates (p = 0.50), blastocyst rates (p = 0.75), and the rate of good quality blastocysts (p = 0.59) between the first and second cycles. The fertilization rate of ICSI was higher in the second cycle than that in the first cycle (69.3 versus 77.5%). Being limited to patients with a low fertilization rate in the first cycle (<60%), the fertilization rate dramatically increased after melatonin treatment (35.1 versus 68.2%). The rate of good quality embryos also increased (48.0 versus 65.6%). An important finding in our study was that oral melatonin supplementation can have a beneficial effect on the improvement of fertilization and embryo quality and this may have occurred due to a reduction in oxidative damage."

http://www.ncbi.nlm.nih.gov/pubmed/24193954

"OBJECTIVE:

To define the therapeutic role of vitamin D in children with moderate to severe bronchial asthma as an adjunct to standard treatment.

METHODS:

Hundred asthmatic children of either sex, attending the respiratory and asthma clinic were enroled in the study. Diagnosis was made on the basis of history and clinical examination. Randomization was done using sealed opaque envelop method. In addition to the treatment as per GINA guidelines, one group received oral vitamin D3 (Cholecalciferol) 60,000 IU per month for 6 mo and the other group received placebo powder in the form of glucose sachet with a double blinded design. Monthly follow up of every patient was done and during every visit change in severity, level of control, Peak expiratory flow rate (PEFR), steroid dosage, number of exacerbations and number of emergency visits were assessed.

RESULTS:

Monthly doses of 60,000 IU vitamin D significantly reduced the number of exacerbations as compared to placebo (p = 0.011). PEFR significantly increased in the treatment group (p = 0.000). Monthly doses of vitamin D significantly reduced the requirement of steroids (p = 0.013) and emergency visits (p = 0.015). Control of asthma was achieved earlier in patients who received monthly vitamin D. Vitamin D significantly reduced the level of severity of asthma patients over 6 mo of treatment (p = 0.016).

CONCLUSIONS:

Vitamin D has a definite role in the management of moderate to severe persistent bronchial asthma as an adjunct to standard treatment.

Lots of these there.

60 posted on 09/08/2014 8:54:38 PM PDT by Black Agnes
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To: freedumb2003

Oh, and the point of my post about the heparin was this.

The whole gist of this article was ‘oh those dirty dirty vitamins and they don’t do anything anyway’.

As it turns out, EVERYTHING from China is suspect. Including most of our pharmaceuticals. Including vaccines. And the FDA has stated it’s not their job to test or inspect any of these. So outlawing those ‘dirty vitamins’ because ‘they don’t do any good anyway’ doesn’t eliminate the problem of Chinese poison in the pill bottles.


63 posted on 09/08/2014 8:57:12 PM PDT by Black Agnes
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