Posted on 05/13/2007 8:21:37 AM PDT by mission9
I think what Hoagland beelieves in is his bank account balance. He’s an opportunist and a con-artist.
At the link in my post, aren’t the references the sources you wanted to see?
Resources
Pesticides Literature Review. April 23, 2004
By Dr. Margaret Sanbord, Dr. Donald Cole, Dr. Kathleen Kerr, Dr. Cathy Vakil, Dr. Luz Helena Sanin, Dr. Kate Bassil. available at: http://www.ocfp.on.ca/english/ocfp/communications/publications/default.asp?s=1#EnvironmentHealth
Pesticides and Human Health
This is an essential resource for physicians and other health providers for understanding the chronic health impacts of pesticides.
Written by Gina Solomon, M.D., M.P.H., Oladele Ogunseitan, Ph.D., M.P.H., and Jan Kirsch, M.D., M.P.H. — and reviewed by nine health professionals — this extensive review study, culled exclusively from peer-reviewed published medical data, delivers a sobering and overwhelming diagnosis that public health institutions, regulators and policymakers are largely unaware of pesticides’ many deleterious effects.
http://www.psrla.org/pesthealth.htm
Reigert,J.R. and J.R.Roberts. 1999. Recognition and Management of Pesticide Poisonings, Fifth Edition. U.S. Environmental Protection Agency and Briggs, S.A. 1992. Basic Guide to Pesticides: Their Characteristics and Hazards
Leiss, J., Savitz D. 1995. Home pesticide use and childhood cancer; a case control study. Am J Public Health 85:249-52 and Daniels O., Savitz D. Pesticides and childhood cancers. Environ Health Perspect 105(10).
Cox C. 1995. Dicamba. J Pesticide Reform 14(1). and Morrison, HI et al. 1992. Herbicides and cancer. J Natl Cancer Inst: 84 (24) 1866-8.
Dick J. et al. 1997. Pesticides and cancer. Cancer Causes and Control 8:420-43; and Smith, JG and Christophers, AJ. 1992. Phenoxy herbicides and chlorophenols: a case control study on soft tissue sarcoma and malignant lymphoma. Br J Cancer 65 (3): 442-48; and Ma X. et al. 2002 Critical windows of exposure to household pesticides and risk of childhood leukemia, Environ Health Perspect 110(9):955-60; and Hardell L, Eriksson M, Nordstrom M. 2002. Exposure to pesticides as risk factor for non-Hodgkin’s lymphoma and hairy cell leukemia: pooled analysis of two Swedish case-control studies. Leuk Lymphoma May;43(5):1043-9
Daniels JL, et al. 2001 Residential pesticide exposure and neuroblastoma. Epidemiology. 12(1):20-26.
Van Der Gulden et al. 1996. Farmers at risk for prostate cancer. Br J Urology 77 (1): 6-14.
Zahm, SM and Ward, MH. 1998 Pesticides and Childhood Cancer. Environ Health Perspect 106(Suppl 3):893-908
Brender, JD, Suarez, L. 1990 Paternal occupation and encephaly. Am J Epidemiol. 11:517-21. and Sever LE et al. 1997. Reproductive and developmental effects of occupational pesticide exposure: the epidemiological evidence. Occupational Medicine; State of the Art Reviews. 12 (2): 303-25.
Guillette, EA et al. 1998. An anthropological approach to the evaluation of preschool children exposed to pesticides in Mexico. Environ Health Perspect. 106: 347-53.
Ecobichon D. 1994. Organophosphorus ester insecticides. In: Pesticides and Neurological Diseases (Ecobichon DJ, Joy RM, eds). CRC Press, Boca Raton, FL; pp 71-250.
Voccia,I et al. 1999. Immunotoxicity and pesticides: a review. Toxicol Ind Hlth. 15: 119-32.
Muir, T and Zegarac, M. 2001 Societal Costs of Exposure to Toxic Substances: Economic and Health Costs of Four Case Studies That Are Candidates for Environmental Causation. Environmental Health Perspectives Volume 109, Supplement 6.
Prepared by Meg Sears MEng, PhD - Please address concerns or comments to healthyottawa@hotmail.com
Environmental Working Group. Body Burden http://www.ewg.org/reports/bodyburden/es.php and “The Pollution in Newborns”. http://www.ewg.org/reports/bodyburden2/
Centers for Disease Control, USA. National Report on Human Exposure to Environmental Chemicals. http://www.cdc.gov/exposurereport/ (July 27, 2005)
United States Veteran Affairs. VA’s Guide on Agent Orange Claims. http://www.vba.va.gov/bln/21/benefits/Herbicide/AOno3.htm (June 13, 2005)
Agency for Toxic Substances and Disease Registry. USA. Toxicological Profile for Polycyclic Aromatic Hydrocarbons (PAHs). August 1995 available via http://www.atsdr.cdc.gov/toxprofiles/tp69.html
Sulentic CE, Holsapple MP, Kaminski NE. Putative link between transcriptional regulation of IgM expression by 2,3,7,8-tetrachlorodibenzo-p-dioxin and the aryl hydrocarbon receptor/dioxin-responsive enhancer signaling pathway. J Pharmacol Exp Ther. 2000 Nov;295(2):705-16.
U.S Department of Health and Human Services – Agency for Toxic Substances and Disease Registry. Toxicological Profile for Chlorinated Dibenzo-p-dioxins. Dec. 1998. available via http://www.atsdr.cdc.gov/toxprofiles/tp104.html
De la Rosa P, Barnett JB, Schafer R. Loss of Pre-B and Igm+ B Cells in the Bone Marrow After Exposure to a Mixture of Herbicides. J Toxicol Env Health. 2003;66(24):2299-2313.
Giesy JP, Kannan K. Dioxin-like and non-dioxin-like toxic effects of polychlorinated biphenyls (PCBs): implications for risk assessment. Crit Rev Toxicol. 1998;28(6):511-69.
Nilsson CB, Hakansson H. The retinoid signaling system—a target in dioxin toxicity. Crit Rev Toxicol. 2002;32(3):211-32.
Schreinemachers D. Mortality from Diabetes Mellitus and associated diseases in four U.S. wheat-producing states. Epidemiology. 2004;15(4)S182.
Sanbord M, Dr. Donald Cole, Dr. Kathleen Kerr, Dr. Cathy Vakil, Dr. Luz Helena Sanin, Dr. Kate Bassil. Pesticides Literature Review. April 23, 2004 available at: http://www.ocfp.on.ca/english/ocfp/communications/publications/default.asp?s=1#EnvironmentHealth
Daniels JL, Olshan AF, Teschke K, Hertz-Picciotto I, Savitz DA, Blatt J, Bondy ML, Neglia JP, Pollock BH, Cohn SL, Look AT, Seeger RC, Castleberry RP. Residential pesticide exposure and neuroblastoma. Epidemiology 2001 Jan;12(1):20-7
Lerda D, Rizzi R. Study of reproductive function in persons occupationally exposed to 2,4-dichlorophenoxyacetic acid (2,4-D). Mutation Res 1991;262:47-50.
Arbuckle TE, Savitz DA, Mery LS and Curtis KM. Exposure to phenoxy herbicides and the risk of spontaneous abortion. Epidemiology 1999;10,752–760.
Schreinemachers DM. Birth Malformations and Other Adverse Perinatal Outcomes in Four U.S. Wheat-Producing States. Environ Health Perspect 2003;111:1259–1264.
Boddington MJ, Gilman AP, Newhook RC, Braune BM, Hay DJ, Shantora V. Canadian Environmental Assessment Act. Priority Substances List. Assessment Report no. 1: Polychlorinated bibenzodioxins and polychlorinated dibenzofurans. Minister of Supply and Services Canada. Catalogue no. En 40-215/1E. ESBN 0-662-17644-8.
International Programme on Chemical Safety, Environmental Health Criteria 29, 2,4-dichlorophenoxyacetic acid (2,4-D). World Health Organization, Geneva. 1984. http://www.inchem.org/documents/ehc/ehc/ehc29.htm
International Programme on Chemical Safety Environmental Health Criteria 84 2,4-Dichlorophenoxyacetic Acid (2,4-D) - Environmental Aspects. Published under the joint sponsorship of the United Nations Environment Programme, the International Labour Organisation, and the World Health Organization World Health Organization. Geneva. 1989 http://www.inchem.org/documents/ehc/ehc/ehc84.htm).
Pest Management Regulatory Agency. “Re-evaluation of the Lawn and Turf Uses of (2,4-Dichlorophenoxy)acetic Acid [2,4-D]” PACR2005-01, February 21, 2005. http://www.pmra-arla.gc.ca/english/pdf/pacr/pacr2005-01-e.pdf. (February 25, 2005).
City of Ottawa. 2004 year-end report on City of Ottawa pesticide reduction strategy for private property. Ref.No: ACS2005-PGM-POL-0029. http://ottawa.ca/calendar/ottawa/citycouncil/hrssc/2005/05-05/ACS2005-PGM-POL-0029%20English.htm (April 28, 2005)
The Dow Chemical Company. Dioxin reports. TRI Dioxin Reporting (U.S.) http://www.dow.com/commitments/debates/dioxin/tri.htm
Chlorine Council toxic waste reports. Available from http://c3.org
* Imidacloprid is a chlorinated nicotinoid compound, that affects the nervous system.
* Imidacloprid is very toxic to earth worms and bees and is restricted in France because of plummeting bee populations.
* Imidacloprid kills parasitic wasps that control grub larvae. This is counter-productive, setting the stage for repeated, more extensive infestations.
* Imidacloprid is applied to seeds because it acts as a bird repellent. Birds such as starlings and robins eat very large numbers of grubs. Repelling this important natural control would be counter-productive.
* The label stipulates that food crops cannot be planted for a year after imidacloprid application. Thus, two growing seasons would have elapsed before harvest. It is not being proposed that our children be afforded the protection of two growing seasons before exposure to turf that has been treated with imidacloprid.
This is the site that is referenced in that report:
http://www.ocfp.on.ca/English/OCFP/Communications/CurrentIssues/Pesticides/default.asp
Ottawa College of Family Physicians.
I just looked at some of these links, looks like the correct references.
>>>>Effects of sub-lethal imidacloprid doses on the
homing rate and foraging activity of honey bees...<<<
Of the neonicotinoids, imidacloprid is the most toxic to birds and fish. Both imidacloprid and thiamethoxam are highly toxic to honey bees.
http://www.freerepublic.com/focus/chat/1990507/posts?page=5922#5922
UNDIAGNOSED DIE-OFF, APIS - UK: PESTICIDES SUSPECTED
****************************************************
A ProMED-mail post
http://www.promedmail.org
ProMED-mail is a program of the
International Society for Infectious Diseases
http://www.isid.org
[1]
Date: 1 Oct 2008
Source: Organic in Form [edited]
http://www.organicinform.org/newsitem.aspx?id=3D588
Call to DEFRA to save bees
The Soil Association has today written to Hilary Benn, DEFRA
Secretary of State, urging him to ban the products in the UK with
immediate effect. There is worldwide concern at widespread,
unexplained and devastating deaths of honey bees over the last 2
years. Bee keepers have reported potentially catastrophic loss of
bees from their hives ranging anywhere from 30-90 percent. Britain’s
beekeepers have reported that close to one in 3 hives have failed to
make it through last winter. This “Colony Collapse Disorder” (CCD) is
not just a problem for beekeepers and farmers, but for consumers as
well, since bee pollination is essential for crop production.
The products implicated in bee deaths, clothianidin, imidacloprid,
fipronil and thiamethoxam, are approved to kill insects on a wide
range of crops in the UK including very widely grown oilseed rape,
barley, and sugar beet. They are also cleared for use in ornamental
plant and hop production.
Peter Melchett, Soil Association Policy Director, says, “It is
typical of the lax approach to pesticide regulation in the UK that we
look like being one of the last of the major farming countries in the
EU to wake up to the threat to our honey bees and ban these nasty
sprays. We want the Government to act today to remove this threat to
Britain’s honey bees.”
Since their introduction by Bayer CropScience in the USA in 2003,
these neonicotinoid products have been linked to the devastating loss
of millions of honey bees in a number of countries. Germany banned
the pesticides after beekeepers in the Baden-Wurttenberg region
reported that two thirds of their bees died in May [2008] following
the application of clothianidin (Poncho Pro). In 1995 beekeepers in
North Dakota took Bayer to court when a third of their bees were
killed by imacloprid. In France, a third of the honey bee population
was killed after widespread use of imidacloprid.
Organic farming relies on a number of techniques to avoid the use of
sprays that kill insects, including not growing the same or similar
crops every year, and encouraging natural predators of insect pests
(like wild birds, ladybirds and lacewings).
—
Communicated by
ProMED-mail Rapporteur Mary Marshall
******
[2]
Date: Tue 30 Sep 2008
Source: Getwokingham - The Wokingham Times [edited]
http://www.getwokingham.co.uk/lifestyle/food_and_drink/s/2036346_bees_disaster_threatens_the_honey_harvest
Bees disaster threatens the honey harvest
In the UK, Stuart Bailey, chairman of Rowse Honey, said recently: “By
Christmas [2008] there will be no English honey in the supermarkets. I’m
absolutely sure, because we’re just not getting the honey in from the
beekeepers. So we will have to import more in from abroad. That’s
tragic. Imagine if that were a permanent scenario: if there are no
British honey-bees, there’ll be no British honey on the shelves.”
Concerned what the dying bees mean for our planet, online discussion
boards warn: “The bees are showing us something here, it is a clear warning.”
Another says: “For many fruit crops, bee loss is a big, big deal.
Gardeners and those with backyard landscapes, please grow bee habitat,
and do it without pesticides, both for honeybees and for native bees.”
Should we be worried? No, says Jon Davey, chairman of Reading and
District Beekeepers’ Association, but the situation is serious. In
his view, the decline of the nation’s — not to mention the world’s —
bee population is something Mother Nature can rectify, with a little
help from her faithful servant, the apiarist. “Bees go back to the
age of the dinosaur. We don’t know why some things happen,” he says.
For example, there are a lot of bumble-bees around this year. We don’t
know why. “Why honeybees are disappearing, who knows? It’s not good
but I should think the bees will survive providing there are
beekeepers to help them.”
Problem is, wild bees producing hives of amber nectar in the woods
are long gone in the UK, rather dating AA Milne’s honey-scoffing bear
Winnie the Pooh [published in the 1930s], says Davey.
Which means we are reliant on beekeepers because not only do bees
supply us with nutritious honey, they form an integral link in the
ecosystem.
Crops such as wheat are pollinated by the wind but apples, pears,
raspberries, cherries, strawberries, blackcurrants, broad and runner
beans and oilseed rape need bees for survival.
The largest threat to bees are varroa mites, a parasite introduced to
Europe and the USA when docile bees were imported from Thailand 15 years ago.
Beekeepers, along with providing secure housing, help bees deal with
the mite “with varying success,” says Davey, and make sure they are
healthy. But add to this 2 dismal summers, meaning bees have not
produced enough honey to keep them going over the winter, and the
situation becomes more serious.
Berkshire beekeepers — most of who are amateur or semi-professional
— do their bit by attending seminars such as the disease recognition
and varroa management day held by regional bee inspector Ian Homer.
Thankfully, most members are dedicated to what is seen in this
country as a left-field hobby.
Davey is committed more than most, for it’s his number that gets handed
out by Reading Borough Council when people call in to report errant
beehives in roofs and such like.
But to solve the problem of our depleted bee population, more
research is needed, says Davey. There is no doubting bee-keepers’
passion for providence (Davey started to keep bees as a solution to
runner beans which refused to germinate), but without more knowledge,
they are limited in what they can do.
There is no doubt British honey will be in short supply by Christmas
[2008], but Davey, who used to work in the trade, says it is highly
unlikely shops will have none at all, because Rowse, Hartley’s and
Gale’s — the big 3 British suppliers — import it from all over the
world, then blend and package it for supermarkets.
However, the world’s big producers of honey, the USA and Argentina,
have also suffered large losses. The former dropped its yield by 36
percent on last year due to colony collapse disorder (CCD), a
mysterious disappearance linked to the blood-sucking varroa mite,
lethal viruses, malnutrition, pesticides, and a lack of genetic
diversity. And in Argentina there was a 27 percent decrease in the
harvest, boosting the price of honey by up to 60 percent.
You can expect to pay around GBP 3 [USD 5.30] for a jar of Berkshire
honey, a creeping-up in price partly the result of the bee crisis but
largely due to the European ban on the import of Chinese honey in 2002.
In the UK, a survey by the British Beekeepers’ Association (BBKA)
revealed that nearly one in 3 of the 240 000 honey-bee hives did not
survive this winter and spring.
BBKA president Tim Lovett says: “Average winter bee losses due to
poor weather and disease vary from between 5 and 10 percent, so a 30
percent loss is deeply worrying. This spells serious trouble for
pollination services and honey producers.”
BBKA is calling on the Government to pour GBP 8 million [USD 14 145
647] over 5 years into researching honey-bee losses and improving bee
health, small change if you consider the value of bees to pollination
— the boost in crop production — was GBP 165m [USD 291 753 971] in
the UK. Currently, GBP 200 000 [USD 353 641] a year is spent on
research and Defra [UK Department for Environment, Food and Rural
Affairs] allocates GBP 1.3 million [USD 2 298 667] for bee health. The
only response to those calls has come from Rowse, which earlier this
month committed GBP 100 000 [USD 176 820] for honey-bee health
research over the next 3 years.
[Byline: Alison Hepworth]
—
Communicated by:
ProMED-mail Rapporteur Susan Baekeland
[The decline in bees, whether from colony collapse disorder, any of a
number of chemicals, or from mites, is taking a serious toll on
pollination and may affect a variety of food crops. - Mod.TG]
[A map of the United Kingdom is available at:
http://www.lib.utexas.edu/maps/cia08/united_kingdom_sm_2008.gif
- CopyEd.EJP]
[see also:
Colony collapse disorder, apis - Germany: chemical ban 20080613.1868
2007
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