Humans can’t survive without ample healthy cholesterol. Cells require healthy cholesterol for renewal and food. The liver creates cholesterol from lipoproteins and fats, either stored or ingested.
When a liver gets too much sugar it starts producing attenuated amounts of cholesterol, mostly compact low density lipoprotein (VLDL). Because production is attenuated the blood fills with unused fat in the form of triglycerides. So high levels of triglycerides and VLDL’s are bad indicators.
The same glycation which causes liver attenuation hardens artery walls. Artery walls become britt!e, and crack when the heart pumps. VLDL particles build up in the cracks until they cause blockages.
Statins are ineffective, and have been associated with cancer. Stop ingesting carbohydrates. Replace your carbohydrates with fats.
Excellent post.
I was just prescribed a statin because of inherited high levels of lp(a).
Because I don’t have high cholesterol and my good cholesterol is great, I’m kind of nervous about taking it. When I questioned it as statins don’t lower lp(a) my cardiologist consulted with a cardiologist at Mayo Clinic and they confirmed that it is how they treat those with elevated lp(a).
From what I can tell, the idea is that less ldl means there is less for the lp(a) to cling to.
I’m 45 and don’t look like a typical cardiology patient. It’s all because of genetics. I have a heart scan in 2 weeks to see if/how much blockage there is. My younger brother died instantly from a heart attack this fall leaving me as the only one in my immediate family to not have had a massive heart attack.
While I do have the high lp(a), I’m the only one who limits sugar and bad fats in my diet and have done so for the past 14 years. I was prescribed Lipitor yesterday so haven’t taken it yet. I just don’t know what is the right thing to do.
This is actually reversible with high dose vitamin K in conjunction with vitamin D. Vitamin K is best absorbed when taken with fat, as it is fat-soluble.