Posted on 08/08/2023 11:11:51 AM PDT by chickenlips
🔬 The puzzle pieces continue to fall into place, and a significant new puzzle piece dropped last week, in the form of a preprint published to MedRxIV on July 31st, titled “SARS-CoV-2 Uses CD4 to Infect T Helper Lymphocytes.”
Although “just” a preprint, it features no fewer than seventy-seven authors. And it has already gathered three positive peer reviews. Let’s start with their conclusion, and then try to unpack what it means. The study’s admittedly obscure conclusion:
We demonstrated that SARS-CoV-2 spike glycoprotein (S) directly binds to the CD4 molecule, which in turn mediates the entry of SARS-CoV-2 in T helper cells. This leads to impaired CD4 T cell function and may cause cell death. SARS-CoV-2-infected T helper cells express higher levels of IL-10, which is associated with viral persistence and disease severity. Thus, CD4-mediated SARS-CoV-2 infection of T helper cells may contribute to a poor immune response in COVID-19 patients.
The researchers figured this out by first using sophisticated genetic software to analyze covid’s RNA. The results showed that covid’s spike protein potentially interacts with 77 different human tissues. But, after they filtered the results for proteins found on the outside of human cells, which are accessible to the spike, they found only one match: something called CD4 helper cells.
Then the researchers proved that covid does target CD4 cells in vivo, including by infecting previously un-infected blood, and by testing blood samples from recovering covid patients. They were very careful and the result seems pretty well established. So it almost seems like the spike was designed to attack this one particular type of cell. But what is it?
-SNIP-
(Excerpt) Read more at coffeeandcovid.com ...
How about providing an insightful dumbed down summary for us old dummies?
bfl
Maybe that’s what the HIV DNA they spliced in their was for.
Likely the easiest summary...
“the spike was designed to attack”
Well, remember that AIDS patients have lowered immune function? That’s because HIV kills their T-cells, which are kind of the immune system’s jack-of-all-trades weapon. They aren’t specialized in fighting a specific infection, like antibodies, but can help fight many different threats. Once you lose those, the rest of the immune system is not able to respond as effectively to threats it hasn’t encountered before.
I’m glad I wasn’t a gullible guini pig who was suckered into taking this death shot. I’m also glad I’m not a willing victim left to defend those who designed this bio weapon, or to try and attack those who bring the evidence into the open.
I see that you are back to fear-monger for November 2024.
GFY.
You’re an idiot that obviously didn’t read the article! Because based on the content of the article, if I’m fear mongering that it makes you a Fauci apologist.
This was about what they have discovered about the WuFlu virus and how it impacts the immune system. It also questions how much of this was already known by the government.
I heard it out with you about Covid before. You’re a big believer in the clot shot so please keep up with them. Don’t you need another “booster” now?
IOW, SARS-CoV-2 is a setup for something else.
Bkmk
That’s BS. I have never been a advocate for the mRNA vax. I doubted it because it was a technology that had never even gotten out of clinical trials for anything it was tried on, and suddenly its a wonder vax. I held off getting the jab hoping the Novavax would get approved and be safe. I finally broke down and got the J&J shot at the insistence of my family, my cardiologist and after two friends ended up in the ICU. Within a day I was regretting it due to the neurological issues it caused. Fortunately they did eventually go away, but the whole affair confirmed my doubts about the vax.
I’ve had WuFlu and it was nothing compared to the side effects of the vax. I’m not an advocate for the death jab. I’ll leave the vax cheerleading to President Warp Speed.
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