How and why? What was the difference between those who were “programmed for success” and those that were not?
One had a better filing system to FIND the “in case of novel anti-ribosome antibiotic - break out THIS gene” part of the genome?
Couldn't I just show how fantasyland your proposed mechanism is by showing the genome of the initial population had only ONE gene for ribosomes?
So where did that starting population of one with only ONE gene for a ribosome grow into a population where some were “programmed for success” and others were not - apparently with access to several different variations of the ribosome gene?
Saying something doesn't indicate a mechanism. How were some “programmed for success” and how were others programmed for failure? What was the molecular difference between the majority of the population that died and the small part of the population that survived?
It is glaringly apparent to this casual observer that you did not even read betty boop’s post. It is as if you are responding to someone else and some other post entirely! I wonder that anyone has the patience to continue trying to post to you at all. But that’s the betty boop I remember!