Glenn Beck Tells Audience He Might Be Going Blind

Politics Dialy ^

[Scythian]

During a tearful and passionate speech at his "American Revival" meeting in Salt Lake City on Saturday, Fox News host Glenn Beck revealed that he may be going blind.

During his speech (video below), Beck told the audience, "I can't focus my eyes."

Taking a shot at health care reform, Beck said, "I went to the best doctor I could find, while I could still go to the best doctor I can find."

Beck, 46, says he has been diagnosed with macular dystrophy, and joked that the doctor told him, "You could go blind in the next year . . . or you might not."

[EyeGuy]

5.6 is an excellent A1C, but other factors need to be considered, such as whether you are insulin-dependent and the longevity of your DM.

Intermittent and variable focusing issues can be just manifestations of the progression of your presbyopia: simple aging. The ongoing accomodative (focusing) decline is not always predictable and linear.

In the absence of prior retinopathy or other pathology, and with good past blood sugar control, I’m not sure why your optometrist or ophthalmologist wants to see you more frequently than annually.

[gunnyg]

Thomas DiLorenzo has Glenn Beck’s Number....

http://gunnyg.wordpress.com/2010/07/19/gunny-g-thomas-dilorenzo-has-glenn-becks-number/

[kruss3]

Get the word to Beck: alpha lipoic acid, lutein, and iron reduction will do wonders for his condition.

Exp Eye Res. 2010 Jun;90(6):664-78. Epub 2010 Mar 15.
Iron, the retina and the lens: a focused review.
García-Castiñeiras S.

Dept. Ophthalmology, School of Medicine, PO Box 365067, San Juan, PR 00936-5067. sixto.garcia@upr.edu
Abstract
This review is focused on iron metabolism in the retina and in the lens and its relation to their respective age-related pathologies, macular degeneration (AMD) and cataract (ARC). Several aspects of iron homeostasis are considered first in the retina and second in the lens, paying particular attention to the transport of iron through the blood-retinal barrier and through the lens epithelial cell barrier, to the immunochemistry of iron-related proteins and their expression in both the retina and the lens, and to the nature of the photochemical damage caused by UV light on both tissues. A comparative overview of some iron related parameters (total iron, transferrin (Tf), transferrin saturation and total iron binding capacity), in plasma and ocular tissues and fluids of three animal species is also presented. Based on results selected from the literature reviewed, and our own results, a scheme for the overall circulation of iron within and out of the eye is proposed, in which, (i) iron is pumped from the retina to the vitreous body by a ferroportin/ferroxidase-mediated process at the endfeet of Müller cells, (ii) vitreal Tf binds this iron and the complex diffuses towards the lens, (iii) the iron/Tf complex is incorporated into the lens extracellular space probably at the lens equator and moves to the epithelial-fiber interface, (iv) upon interaction with Tf receptors of the apical pole of lens epithelial cells, the iron/Tf complex is endocytosed and iron is exported as Fe(3+) by a ferroportin/ferroxidase-mediated process taking place at the basal pole of the epithelial cells, and (v) Fe(3+) is bound to aqueous humor Tf and drained with the aqueous humor into systemic blood circulation for recycling. The proposed scheme represents an example of close cooperation between the retina and the lens to maintain a constant flow of iron within the eye that provides an adequate supply of iron to ocular tissues and secures the systemic recycling of this element. It does not discount the existence of additional ways for iron to leave the eye through the blood-retinal barrier. In this review both AMD and ARC are recognized as multifactorial diseases with an important photoxidative component, and exhibiting a remarkable similitude of altered local iron metabolism. The epidemiological relationship between ARC and ferropenic anemia is explained on the basis that hepcidin, the hormone responsible for the anemia of chronic inflammation, could paradoxically cause intracellular iron overload in the lens by interfering with the proposed ferroportin/ferroxidase-mediated export of iron at the basal side of the anterior lens epithelium. Other authors have suggested that a similar situation is created in the retina in the case of AMD. Copyright 2010 Elsevier Ltd. All rights reserved.

PMID: 20230820 [PubMed - indexed for MEDLINE]

[dangerdoc]

I’m a preventive medicine guy, why in the world would somebody name an anterior chamber diseases macular?

It seems to me like naming a dental disease after the rectum.

I’ve never come across the diagnosis of macular dystrophy in my practice, I may need to crack a book later.

[dangerdoc]

For what it is worth Vitelliform macular dystrophy does affect the macula.

[kimmie7]

That is uncalled for, and smells like the stuff those at the DUmp shovel.

[jobim]

Gurn's Argument Technique (GAT):

step 1: Approach opponent (aka douchebag do do head)
step 2: Stick one's tongue out as far as as possible
step 3: place thumbs at ear openings, and with hands spread open, move fingers up and down in a waving motion
step 4: say "Na, Na, Na, Na, Na" loudly

Then run to Momma's house, down to the basement, where warm milk, cookies, and computer are located.

[MWestMom]

You’re right—the libtards will throw a big party over this.

____________________________________________________________

As if blindness would slow him down.

[jazminerose]

Exactly. They would enjoy watching Beck & his family suffer but of course, he won’t be silenced.

Meanwhile, prayers up.

[EyeGuy]

Yeah, I tend to agree.

Without researching it, and thus shooting from the hip, I seem to remember the “macular” terminology in regards to the corneal dystrophy has to do with the way it is physically manifested; the appearance of the pathology. There is no structure or layer of the cornea called the macula.

I’ll have to ask ther retina guys I know, about the rarer retinal “macular dystrophy”. If anything, I remember that as being part of an array of congenital macular dieseases including the macular dysplasias.

[Yosemitest]

Let me help you.

Exp Eye Res. 2010 Jun; 90(6):664-78. Epub 2010 Mar 15.
Iron, the retina and the lens: a focused review.
García-Castiñeiras S.
Dept. Ophthalmology,
School of Medicine,
PO Box 365067,
San Juan, PR 00936-5067.
sixto.garcia@upr.edu

Abstract
This review is focused on iron metabolism
in the retina and in the lens
and its relation to their respective age-related pathologies, macular degeneration (AMD) and cataract (ARC).
Several aspects of iron homeostasis are considered
first in the retina and second in the lens,
paying particular attention to
the transport of iron through the blood-retinal barrier and through the lens epithelial cell barrier,
to the immunochemistry of iron-related proteins and their expression in both the retina and the lens,
and to the nature of the photochemical damage caused by UV light on both tissues.
A comparative overview of some iron related parameters
(total iron, transferrin (Tf), transferrin saturation and total iron binding capacity),
in plasma and ocular tissues and fluids of three animal species is also presented.
Based on results selected from the literature reviewed, and our own results,
a scheme for the overall circulation of iron within and out of the eye is proposed,
in which,
(i) iron is pumped from the retina to the vitreous body by a ferroportin/ferroxidase-mediated process at the endfeet of Müller cells,
(ii) vitreal Tf binds this iron and the complex diffuses towards the lens,
(iii) the iron/Tf complex is incorporated into the lens extracellular space probably at the lens equator and moves to the epithelial-fiber interface,
(iv) upon interaction with Tf receptors of the apical pole of lens epithelial cells,
the iron/Tf complex is endocytosed
and iron is exported as Fe(3+) by a ferroportin/ferroxidase-mediated process taking place at the basal pole of the epithelial cells,
and (v) Fe(3+) is bound to aqueous humor Tf and drained with the aqueous humor into systemic blood circulation for recycling.
The proposed scheme represents an example of close cooperation between the retina and the lens
to maintain a constant flow of iron within the eye
that provides an adequate supply of iron to ocular tissues
and secures the systemic recycling of this element.
It does not discount the existence of additional ways for iron to leave the eye through the blood-retinal barrier.
In this review
both AMD and ARC are recognized as multifactorial diseases
with an important photoxidative component,
and exhibiting a remarkable similitude of altered local iron metabolism.
The epidemiological relationship between ARC and ferropenic anemia is explained
on the basis that hepcidin,
the hormone responsible for the anemia of chronic inflammation,
could paradoxically cause intracellular iron overload in the lens
by interfering with the proposed ferroportin/ferroxidase-mediated export of iron
at the basal side of the anterior lens epithelium.
Other authors have suggested
that a similar situation is created in the retina in the case of AMD.

Copyright 2010 Elsevier Ltd. All rights reserved.

PMID: 20230820 [PubMed - indexed for MEDLINE]

---

My brother suffered from a similar eye problem about 5 years ago.
His girlfriend did allot of research, starting with www.macular-dystrophy.com, and had my brother start taking

Iron supplements, lutein supplements, increased water intake, and increased dark leafy greens.

It improved his eyes and after 4 years the problem went disappeared.
My brother complained of his eyes watering allot while he was on it, but he's happy that his eyesight improved, and recommends Lutein to anyone who's having vision problems.

Check out Lutein and Age related Macular Degeneration (AMD).

And pray for Glenn Beck and his family.

[Gurn]

Some nobody on the internet obsessing over some moment he must have witnessed, where the existence of a turned commie symp that died before he was born slipped somebody's mind, means nothing about the intelligence or standing of the object of said nobody's obsession.

We can stipulate that I'm a nobody on the internet. I'm sure you're much more substantive. But your characterization of Chambers is objectively inaccurate and trivializes the man's importance.

You, however, know way more about him than that crybaby doofus on Fox News, WHO IS ENOUGH OF A MORON TO THINK THE GARDEN OF EDEN WAS IN MISSOURI.

[cowboyusa]

One differnce, Sam Adams advoacated physical resistance to tyrany.

[cowboyusa]

One difference, Sam Adams advocated physical resistance to tyrany.